Focus on understanding key concepts, not memorizing sample questions.
1. Where may you see reciprocal ECG changes (ST depression) in an inferior MI?
2. When you see isolated ST segment depression in leads V1 and V2, you should repeat the 12 lead to view which additional ECG leads?
3. Where is ECG lead V4R placed?
4. How do you calculate a mean arterial pressure (MAP)?
5. Why is pulse pressure narrow in early shock?
6. What hemodynamic changes are seen in the early hyperdynamic phase of septic shock?
7. Is the pulmonary artery obstructive pressure (PAOP) increased or decreased in left ventricular failure?
8. Is systemic vascular resistance (SVR) increased or decreased in hypovolemic shock?
9. Is systemic vascular resistance (SVR) increased or decreased in distributive shock?
10. Is the ejection fraction normal or low in hypertrophic cardiomyopathy?
11. Why are the right atrial pressure (RAP), pulmonary artery diastolic (PAD), and PAOP equalized in pericardial tamponade?
12. How do positive inotropes increase cardiac output?
13. Why is it recommended to maintain the heart rate at 60-80 bpm in a patient with a dissecting AAA?
14. What acid-base disorder do you see in persistent vomiting?
15. What is the happening when you see a “shark-fin” appearance on the end-tidal CO2 waveform?
16. Why is the patient in neurogenic shock bradycardic?
17. In DKA, when is the insulin infusion discontinued?
18. What causes the dark urine in rhabdomyolysis?
19. Why is creatine kinase (CK) elevated in rhabdomyolysis?
Order the CCRN Study Guide and start studying today. Review a sample of my CCRN "Key Points" and a step-by-step guide to hemodynamic monitoring on my blog posts too.